The mechanism of action was proved to be reversible phosphorylation and dephosphorylation.
其机理亦是通过可逆的磷酸化及脱磷酸化进行的。
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It's known that salicylates like aspirin are able to uncouple oxidative phosphorylation, which might help to explain their involvement in mitochondrial destruction, but it's unclear what relationship exists with viral infection, though there certainly seems to be one.
像阿司匹林这类水杨酸盐化合物能对氧化磷酸化解偶联,即供能通路被阻断,这也许能有助于解释线粒体受损的情况,它与病毒性感染也有关系,但是具体联系还不清楚。