They have typical laboratory findings, including hypokalemia, metabolic alkalosis, hypomagnesemia, and hypocalciuria.
在此篇文章中,我们三个例:一性个案,另两位姐弟。
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Individuals with hyperaldosteronism typically develop hypertension related symptoms like headaches and facial flushing, as well as hypokalemia related symptoms like constipation, weakness, and potentially changes in their heart rhythm.
醛固酮增多症患者通常会出现高血压相关症状,如头痛和面部潮红,以及低血症相关症状,如便秘、乏力与对心律的潜在影响。
All right, as a quick recap, hyperaldosteronism is the chronic, excess secretion of aldosterone from the zona glomerulosa of the adrenal gland, and these high aldosterone levels can lead to hypokalemia, hypernatremia, hypertension, and a metabolic alkalosis.
得!简单回顾一下:醛固酮增多症是指肾上腺球状带长期分泌过多的醛固酮;这些高醛固酮水可导致低血症、高钠血症、高血压与代中毒。
As hypokalemia becomes more severe especially in patients with other heart conditions, the inward current may exceed the outward current resulting in early after depolarization and consequently extra heartbeats prolonged repolarization may also promote re-entrant arrhythmias.
随着低血症变得更加严重,尤其是其他心脏病患者,内向电流可能会超过外向电流,从而导致早期去极化后,因此,额外的心跳延长复极也可能促进折返心律失常。
All that being said, in situations where somebody produces too much aldosterone, like primary hyperaldosteronism, then there's more potassium secretion by the principal cells and excreted, and therefore less potassium is retained, leading to hypokalemia.
说了这么多,在某人产生了太多醛固酮的情况下,比如原发醛固酮增多症,更多的被主细胞分泌并排泄,这意味着更少的被保留,导致了低血症。
Taken together, a relatively high potassium concentration in the principal cell and a relatively low potassium concentration in the lumen, strengthens the potassium gradient and causes a lot of potassium to get secreted and excreted in the urine - leading to hypokalemia.
总之,主细胞中相对高的浓度和管腔中相对低的浓度,强化了的浓度梯度,造成了许多被分泌和排泄入液,导致低血症。