Genetic testing for HLA-B27 can be done to help confirm the diagnosis.

检测到HLA-B27基因能够协助确诊。

Each of us have different HLA profiles. The study is elegant science.

我们每个人的HLA情况都不同。这是优雅的科学。

A set of genes called HLA, for instance, are involved in running the immune system.

例如，一组被称为HLA的基因参与了系统的运行。

Turns out, these women also share a mutation in the gene called HLA-B27 that the others lack.

原来，这些女性在HLA-B27基因上有突变，而别人没有。

This adaptive arm can recognise the signature of " selfness" provided by an individual's HLA proteins.

这种自适应臂可以识别个体 HLA 蛋白提供的“自我”特征。

Everyone's HLA profile is slightly different, and reveals which kinds of diseases we have immunity to.

每个人的 HLA 概况略有不同，揭示了我们对哪些病具有力。

All right, as a quick recap, ankylosing spondylitis is an autoimmune process, associated with the HLA-B27 gene, which attacks vertebral joints.

好，简单回顾下：强直性脊柱炎是一种与HLA-B27基因有关的自身病，会导致脊柱关节受累。

HLA is a group of proteins that play a role in coding for some of our immune response.

HLA 是一组蛋白质， 在编码我们的某些应中发挥作用。

In some studies, single people have been found to prefer the BO of HLA dissimilar individuals.

在一些中， 发现单身人士更喜欢 HLA 不同个体的 BO。

One really important group of genes involved in regulation of the immune response is the human leukocyte antigen system, or HLA system.

一组涉及调节应的重要基因叫作人体白细胞抗原系统，或HLA系统。

Unlike with blood transplants, there are thousands of HLA types, and even siblings and close relatives may not have compatible bone marrow.

与输血不同HLA（人类白细胞抗原）有几千种，甚至患者与兄弟姐妹、近亲的HLA都不一定能相容。

Lack of HLA proteins does, however, bring a cell to the attention of immunity's other arm, the innate system.

然而，缺乏 HLA 蛋白确实会引起系统的另一个分支——先天系统的注意。

Disrupting beta cells' HLA expression is not the only possible approach to deflecting the adaptive immune system.

破坏 β 细胞的 HLA 表达并不是转移适应性系统的唯一可能方法。

Most individuals with ankylosing spondylitis have the gene HLA-B27, which is a gene that encodes for a specific type of MHC class I molecule.

许多强直性脊柱炎患者都携带HLA-B27基因，该基因能编码一种特定的MHC Ⅰ类分子。

The first part of Dr Schrepfer's approach is therefore to prevent the production of HLA proteins in laboratory-grown beta cells destined for transplant.

因此，Schrepfer 博士方法的第一部分是阻止用于移植的实验室培养的 β 细胞中产生 HLA 蛋白。

However, the most comprehensive study of its kind found no link between BO preference, HLA dissimilarity and chosen partners in 3,700 married couples.

然而， 同类中最全面的发现，在 3, 700 对已婚夫妇中， BO 偏好、 HLA 差异与选择的伴侣之间没有联系。

So, specifically with autoimmune hepatitis, there seems to be some sort of connection between these particular " self-proteins" , the HLA-DR, and mounting an attack against your own liver.

具体来说，对于自肝来说，HLA-DR这些特殊的“自身蛋白”似乎与系统攻击自己的肝脏之间有某种联系。

If the immune system comes across non-self HLA proteins, it recognises the cells displaying them as interlopers and attacks, using shock troops called killer T-cells and antibodies.

如果系统遇到非自身 HLA 蛋白，它就会将显示这些蛋白的细胞识别为闯入者，并使用称为杀伤性 T 细胞和抗体的突击部队发起攻击。

Genetic risk factors include being a woman and having genes that encode a specific type of immune molecule called HLA-DR2 which is used to identify and bind to foreign molecules.

基因危险因素有身为女性和一种编码特殊类型的分子——HLA-DR2的基因序列（常常识别和结合外源分子）。

Its troops are called NK (natural killer) cells and macrophages, and one of the red flags it reacts to is an absence of any sort of HLA.

它的部队被称为 NK（自然杀伤）细胞和巨噬细胞，它作出应的危险信号之一是任何类型的 HLA 的缺失。